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When blood pressure is low blood vessels tighten to increase pressure
When blood pressure is low kidneys release renin
Renin breaks down angiotensinogen into angiotensin I
Angiotensin I converted to Angiotensin II in lungs
ACE inhibitors act in the lungs
ACE inhibitor blocks enzyme from converting angiotensin I to angiotensin II
ACE breaks down bradykinin
Increased bradykinin can lead to cough
Angiotensin II increases squeezing on blood vessels
Angiotensin II reduction decreases aldosterone
Aldosterone increases reabsorption Sodium
Aldosterone gets rid of potassium
Less aldosterone causes potassium to increase
ACE inhibitors can increase potassium levels (hyperkalemia)
ACE inhibitors lower risk for stroke and heart attack
*Angiotensinogen, ACE, aldosterone
ACE inhibitors
Angiotensin II, Enzyme, Lung
Angiotensin II, Enzyme, Lung
Aldosterone
Potassium, Potassium, Sodium
Potassium, Potassium, Sodium
Angiotensin I
Enzyme
Enzyme
Angiotensin II
Aldosterone, Blood vessels
Aldosterone, Blood vessels
Angiotensinogen
Angiotensin I
Angiotensin I
Blood Pressure
Kidney
Kidney
Enzyme
Angiotensin II, bradykinin
Angiotensin II, bradykinin
Kidney
Renin
Renin
Renin
Angiotensinogen
Angiotensinogen
What do ACE inhibitors block? Enzyme Angiotensin converter enzyme
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What do ACE inhibitors prevent production of? Angiotensin II
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What does angiotensin I convert into? Angiotensin II Angie 1.0 to 2.0
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What decreases with ACE inhibitors? Aldosterone angiotensin II stimulates aldosterone
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What decreases with ACE inhibitors? Sodium aldosterone increases sodium reabsorption
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